DEPOPULATION - what elites want -

[Len Colby]

LOL , any evidence:

-the MIBH are behind this?

-this has lead to lower birth rates?

-it effects poor people or countries more than wealthy ones? The source article seems to suggest the opposite:

Sperm count: How to boost it

1.

Wear loose underwear – to make healthy sperm the testicles need to be below body temperature.

2.

Eat food low in saturated fat.

3.

Avoid smoking, drinking, using drugs and becoming obese.

4.

Reduce exposure to industrial chemicals such as those used in making plastics – they can mimic the female hormone oestrogen countering male hormones.

5.

Protect women in pregnancy – there is growing evidence that falling sperm counts may stem from effects in the womb.

6.

Avoid anti-depressants – in rare cases they can cut sperm counts

.

http://www.independent.co.uk/news/science/scientists-warn-of-sperm-count-crisis-8382449.html

Health Statistics > Obesity (most recent) by country

http://www.nationmaster.com/graph/hea_obe-health-obesity

[Greg Burnham]

deleted

Edited December 31, 2012 by Greg Burnham

[Steven Gaal]

CORPORATIONS FIGHT TO HAVE THINGS THAT LOWER SPERM COUNT

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Plastic Chemical BPA Linked to Lower Sperm Count & Quality

By Jennifer Welsh | October 29, 2010 4:39 pm

A new study of 218 Chinese men found that even low levels of the controversial plastics chemical bisphenol A (BPA) can lower sperm quality and count.

For the study, which was published in the journal Fertility and Sterility, researchers noted the participants’ sperm quality and urine BPA levels over five years. When compared to participants without detectable levels of the chemical, men with BPA in their urine were three times more likely to have low quality sperm.

“This adds additional human evidence that BPA is bad,” said [the study's first author] De-Kun Li…. “The general public should probably try to avoid exposure to BPA as much as they can.” [Washington Post]

That’s a tough order, because BPA is all over the place. It’s found in everything from sports equipment to medical devices to the plastic lining in canned foods.

Li’s previous studies have shown sexual effects of high levels of BPA, including inducing impotence in male factory workers exposed to it. Those studies were done with men exposed to about 50 times as much BPA as the average U.S. man, so the results might not apply to your average Joe.

However, in the current study Li argues that the chemical didn’t only have a detrimental effect on individuals with sky-high BPA levels. Even participants with low levels, comparable to the average American, had diminished sperm quality and count.

“The higher your exposure, the lower your sperm quality is,” he said. [Washington Post]

Much of the controversy about BPA has focused on the question of what level of BPA exposure is safe. This study adds weight to the argument that the chemical is hazardous to everyday people.

“Evidence has indicated that for the past few decades, sperm counts have been declining in some human populations–and that this might be related to exposures to endocrine-disrupting chemicals such as BPA is very reasonable,” [Gail] Prins said. “I strongly believe that the U.S. should take measures to reduce the use of this chemical, since levels build over time.” [Live Science]

Canada must be feeling smug, since the country officially labeled the chemical as toxic earlier this month, making it easier to ban it and reduce environmental exposure to the chemical.

Related content:

Discoblog: Warning All Competitive Male Cyclists: Less than 5% of Your Sperm May Be Normal

80beats: Study Finds BPA in Store Receipts; Health Effects as Yet Unclear

80beats: BPA-Heart Disease Link Confirmed, But Levels in People Have Declined

80beats: BPA Won’t Leave Public-Health Conversatio–or Your Body

80beats: More Bad News on BPA: Linked to Heart Disease and Diabetes in Humans

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J Hum Reprod Sci. 2010 Sep-Dec; 3(3): 148–152.

doi: 10.4103/0974-1208.74159

Effect of duration of fluoride exposure on the reproductive system in male rabbits

Naresh Kumar, Sushma Sood, B Arora,¹Manjeet Singh, and Beena

Department of Physiology, Pt B.D. Sharma Post Graduate Institute of Medical Sciences, Rohtak, Haryana, India

¹Department of Pathology, Pt B.D. Sharma Post Graduate Institute of Medical Sciences, Rohtak, Haryana, India

Address for correspondence: Dr. Naresh Kumar, 11 J/3 Medical Campus, Post Graduate Institute of Medical Sciences, Rohtak, Haryana - 124 001, India. E-mail: drnaresh10@gmail.com

Author information ► Article notes ► Copyright and License information ►

Received April 19, 2010; Revised October 18, 2010; Accepted November 11, 2010.

Copyright © Journal of Human Reproductive Sciences

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Go to:

Abstract

BACKGROUND AND OBJECTIVES:

Fluorosis has become an endemic problem worldwide. Fluoride has its effect on various organs, including the reproductive system, although there are controversial reports over it. Thus, the present study was designed to study the effect of sodium fluoride (NaF) exposure for different durations

INTRODUCTION

Humans are exposed to sodium fluoride (NaF) from a number of sources, including water, medicines, pesticides, insecticides, fertilizer residues, dental restorative materials, dental products (tooth pastes and mouth rinses), pediatric supplements, beverages prepared with fluoridated water and food.[1] Fluorosis is an endemic public health problem in nearly 22 nations around the world. The World Health Organization (WHO) guideline is that 1.5 ppm of fluoride is the desirable upper limit in drinking water. The magnitude of problem of fluorosis in India is roughly estimated to be of 66.62 million people at risk.[2] The recommended levels of fluoride in drinking water are 0.5–0.8 mg/L. Fluoride levels above 1.5 mg/L may lead to dental fluorosis while levels above 3–6 mg/L during the life time may lead to skeletal fluorosis.[3] Involvement of the reproductive organs due to fluorosis in animals had also been studied extensively. Messar et al., reported that the low levels of fluoride in food rendered mice infertile, while a high-fluoride diet improved their fertility.[3] Chinoy and Sequeira reported that sodium fluoride treatment in mice caused an alteration in the histology of reproductive organs and morphology of sperm and induced biochemical changes.[4] These reports were contradicted by Tao and Suttie, whose experiments showed that fluoride did not play any essential role in reproduction.[5] Because of these controversial reports, we intended to assess the effect of fluoride on the rabbit reproductive system.

DISCUSSION

Animal studies had shown contradictory reports regarding the effect of fluorosis on the reproductive system. Therefore, the present study was performed to establish the link between fluoride and its effect on sperm count, motility, progressive motility, weight of testis, weight of epididymis and histopathology of the testis of age and body weight matched groups.[6]

Effect on sperm count

It was observed that in comparison with the control group, there was a significant decrease in the epididymal sperm count when NaF was given for 30 days to rabbits and a greater decrease when NaF was given for 60 days. Hence, the present study proved that higher the fluoride exposure, higher the effect on sperm counts. Similar results had been observed in rats, mice and rabbits in studies performed earlier.[8–10]

The effect of fluoride toxicity on spermatogenesis could be because fluoride reduces the testosterone levels and by reducing the testicular zinc levels, it impairs angiotensin-converting enzyme (ACE) activity and hence causes inhibition of spermatogenesis.[1] Apart from the direct effect on the levels of testosterone, fluoride inhibits androgen receptor (AR) mRNA expression in Sertoli cells and causes a decrease in AR through which testosterone acts.[11] Wan et al., reported that epidermal growth factor (EGF) and its receptor (EGFR), which plays an important role in male reproductive functions of rat, were significantly decreased in Leydig cells, spermatogonia and spermatocytes on exposure to 68 ppm fluoride for 10 days.[12] Other factors responsible for the arrest of spermatogenesis might be the lack of available proteins necessary for cell division, growth and differentiation of germ cells. In a study, disruption of normal cell cycle and apoptosis was indicated at doses of 200–30 mg/L of fluoride, which may be attributed to the blockage of the G1 phase of the cell cycle. Another mechanism of apoptosis is increased levels of oxidants, which damages the DNA.[13]

Effect on motility

In the present study, there was a significant decrease in sperm motility and progressive motility in the group fed with fluoride 20 mg/kg for 30 days and 60 days as compared with the control. Huang et al., also reported a significant reduction in the sperm motility of mice fed on 100, 200 and 300 mg NaF/L for 8 weeks as compared with the control group.[13] Similar results were shown in rats and mice in many other studies.[9,14–16] One important study demonstrated that human spermatozoa lost their motility in vitro in the presence of 250 mM fluoride within 20 mins of exposure and, similarly, fluoride (30 mM) made the bull sperms immotile within 2 min at 30°C in vitro.[8]

The mechanism by which fluoride affects sperm motility has not been clearly elucidated. However, it has been postulated that fluoride could act directly on the motile apparatus without affecting other metabolic systems.[17] One mechanism could be decline in the fructose level, which provides energy for motility, in the seminal vesicle and vas deferens due to alteration in carbohydrate.[8,17] Fluoride may also act by inhibiting many enzymes: the first mode of action is that fluoride binds with cofactors like Mg, Ca, Zn and Se and thus inhibits glycolysis, respiration and motility of sperms, or it might also form insoluble complexes with magnesium or phosphates in enzymes like enolase and acid and alkaline phosphatase.[17] Another reason for decreased sperm motility was decreased level of androgen carrier proteins involved in sperm motility.[18] Furthermore, structural defects were observed in the flagellum, acrosome and nucleus of spermatids and epididymal spermatozoa of fluoride-treated rabbits (10 mg/kg/day for 18 months) leading to abnormal motility.[19]

Organ weights

In our study, there was a significant decrease in the epididymal weight in Groups II and III as compared with the control while there was a significant decrease in the testicular weight in Group III only, indicating that a higher level of NaF is needed to affect testicular weight than epididymal weight. In other words, we can conclude that fluoride affects epididymal weight earlier than testicular weight.

Similarly, in another study, rabbits fed on fluoride were having a significant decrease in epididymal weight.[20] Also, the weight of the cauda epididymis in fluoride-treated (10 mg/kg for 30 days) mice declined significantly compared with the control groups.[9] However, no significant difference was observed in the mean testicular weights of rats fed on 100 and 300 ppm fluoride for 12 weeks when compared with the control. It might be because of the lower fluoride concentration.[16] Our findings differed from those of Ghosh et al., who reported an increase in the relative testis weight of rats as compared with the control with fluoride treatment (20 mg/kg for 29 days), which might had been due to a compensatory change or due to fluid accumulation in the testis.[21]

Testicular histology

Figures ​Figures11–3 show that longer the fluoride exposure, higher was the damage to the reproductive system.

There was a lack of differentiation and maturation of spermatocytes, with no mature spermatozoa seen in the lumens in Group II [Figure 2] and complete cessation of spermatogenesis in Group III [Figure 3] as compared with normal spermatogenesis in the control group [Figure 1]. Thus, our results were similar to a study that showed that 30 days of treatment with sodium fluoride (10 mg/kg body weight) to mice resulted in sloughing off of the spermatogenic cells in the luminal regions of the seminiferous tubules of the testis, leading to disorganization of their epithelium, which caused a complete absence of spermatogenesis in the testis.[18] Also, direct injection of sodium fluoride (50 µg/50 µl) into the vas deferens resulted in spermatogenic arrest, the absence of spermatozoa in the seminiferous tubules, a decreased sperm count in the cauda epididymis and a decrease in fertility,[22] while Sprando et al., showed that rats fed on a 250 ppm fluoride for 10 weeks showed no distinguishable change in testicular histology from their control group.[1] This might be due to a lower level of fluoride. Susheela et al., in their study on rabbits found that there was disruption of spermatogenic cells in the seminiferous tubules that were degenerated and devoid of spermatozoa. In animals treated for 18 or 29 months, loss of cilia on the epithelial cells lining the lumen of the ductuli efferentes of the caput epididymidis and of the stereocilia on the epithelial cells lining the lumen of the vas deferens was also observed. Similarly, in our study, there was a lack of spermatogenesis, as evident on testicular histology.[10]

Go to:

CONCLUSION

Fluoride definitely has adverse effects on the reproductive functions of male rabbits, and these effects are more pronounced with the increasing duration of exposure.

Go to:

Footnotes

Source of Support: Nil

Conflict of Interest: None declared

Go to:

REFERENCES

1. Sprando RL, Collins TF, Black T, Olejnik N, Rorie J. Testing the potential of sodium fluoride to affect spermatogenesis: A morphometric study. Food Chem Toxicol. 1998;36:1117–24. [PubMed]

2. Susheela AK. Treatise on Fluorosis. 3rd ed. Delhi: Published by Fluorosis Research and Rural Development Foundation; 2006. pp. 1–119.

3. Messer HH, Armstrong WD, Singer L. Fertility impairment in mice on a low fluoride intake. Science. 1972;177:893–4. [PubMed]

4. Chinoy NJ, Sequeira E. Effects of fluoride on the histoarchitecture of reproductive organs of the male mouse. Reprod Toxicol. 1989;3:261–7. [PubMed]

5. Tao S, Suttie JW. Evidence for a lack of an effect of dietary fluoride level on reproduction in mice. J Nutr. 1976;106:1115–22. [PubMed]

6. Chinoy NJ, Sequeira E, Narayana MV. Effects of vitamin C and calcium on the reversibility of fluoride induced alterations in spermatozoa of rabbit. Fluoride. 1991;24:29–39.

7. Saalu LC, Oluyemi KA, Omotuyi IO. Tocopherol (vitamin E) attenuates the testicular toxicity associated with experimental cryptorchidism in rats. Afr J Biotechnol. 2007;6:1373–7.

8. Chinoy NF, Narayana MV, Dalal V, Rawat M, Patel D. Amelioration of fluoride toxicity in some accessory reproductive glands and spermatozoa of rat. Fluoride. 1995;28:75–86.

9. Chinoy NJ, Sharma A. Amelioration of fluoride toxicity by vitamins E and D in reproductive functions of male mice. Fluoride. 1998;31:203–16.

10. Susheela AK, Kumar A. A study of the effect of high concentration of fluoride on the reproductive organs of male rabbit, using light and scanning microscopy. J Reprod Fertil. 1991;92:353–60. [PubMed]

11. Huang C, Yang H, Niu R, Sun Z, Wang J. Effect of sodium fluoride on androgen receptor expression in male mice. Fluoride. 2008;41:10–7.

12. Wan S, Zhang J, Wang J. Fluoride-induced changes in the expression of epidermal growth factor and its receptor in testicular tissues of young male rats. Fluoride. 2006;39:121–5.

13. Huang C, Niu R, Wang J. Toxic effects of sodium fluoride on reproductive function in male mice. Fluoride. 2007;40:162–8.

14. Narayana MV, Chinoy NJ. Reversible effects of sodium fluoride ingestion on spermatozoa of the rat. Int J Fertil Menopausal Stud. 1994;39:337–46. [PubMed]

15. Narayana MV, Chinoy NJ. Effect of fluoride on rat testicular steroidogenesis. Fluoride. 1994;27:7–12.

16. Bataineh HN, Nusier M. Impact of 12-week ingestion of sodium fluoride on aggression, sexual behavior and fertility in adult male rats. Fluoride. 2006;39:293–301.

17. Zakrzewska H, Udala J, Blaszczyk B. In vitro influence of sodium fluoride on ram semen quality and enzyme activities. Fluoride. 2002;35:153–60.

18. Chinoy NJ, Shukla S, Walimbe AS, Bhattacharya S. Fluoride toxicity on rat testis and cauda epididymal tissue components and its reversal. Fluoride. 1997;30:41–50.

19. Kumar A, Susheela AK. Ultrastructural studies of spermiogenesis in rabbit exposed to chronic fluoride toxicity. Int J Fertil Menopausal Stud. 1994;39:164–71. [PubMed]

20. Kumar A, Susheela AK. Effects of chronic fluoride toxicity on the morphology of ductus epididymis and the maturation of spermatozoa of rabbit. Int J Exp Pathol. 1995;76:1–11. [PMC free article] [PubMed]

21. Ghosh D, Das S, Sarkar S, Maiti R, Jana D, Das UB. Testicular toxicity in sodium fluoride treated rats: Association with oxidative stress. Reprod Toxicol. 2002;16:385–90. [PubMed]

22. Chinoy NJ, Rao MV, Narayana MV, Neelakanta E. Microdose vasal injection of sodium fluoride in the rat. Reprod Toxicol. 1991;5:505–12. [PubMed]

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Study finds sperm count decreasing worldwide | Down To Earth

Growing trend of compound sources in air pollution: report .... was a declining trend in sperm counts in USA, Western Europe and many Third World countries.

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Science News

Declining Male Fertility Linked To Water Pollution

Jan. 20, 2009 — New research strengthens the link between water pollution and rising male fertility problems. The study, by Brunel University, the Universities of Exeter and Reading and the Centre for Ecology & Hydrology, shows for the first time how a group of testosterone-blocking chemicals is finding its way into UK rivers, affecting wildlife and potentially humans.

Edited December 31, 2012 by Steven Gaal

[Len Colby]

So have any of these studies be corroborated? Such claims are only considered established if multiple studies point the same way. The authors of the Chinese study acknowledged “these results provide the first epidemiologic evidence of an adverse effect of BPA on semen quality” and that “despite a markedly reduced sample size, the inverse correlation between increased urine BPA levels and decreased sperm concentration and total sperm count remained statistically significant.” Studies with small sample sizes are not considered reliable and even though the study included “men with environmental BPA exposure at levels comparable with those in the U.S population” it was limited to “four regions in China where high exposure to BPA in the workplace existed” correlation does not always mean causation.

But let's assume the authors are correct and BPA exposure leads to decreased sperm quality/counts, the lead author said “the higher your exposure, the lower your sperm quality is.” This implies the effects are proportional, so low exposure levels, i.e. ones typically seen by men who do not work in factories where BPA products are made would lead to small reductions in sperm quality.

The fluoridation study was on rabbits and was published in an 'open access' journal with a minimal impact factor. This suggests the authors were unable to or thought they would be unable to get it publish in a more prominent publication. They acknowledged that “animal studies had shown contradictory reports regarding the effect of fluorosis on the reproductive system”. Have any studies shown such a correlation in humans?

Another problem for you is that water fluoridation is more common in wealthy countries and your theory is that there is a plot to reduce the birth rates in poor ones. As for BPA exposure, other than for factor worker,s is it more common in rich countries or poor ones? Do you have any evidence reduced sperm counts have led to reduced birth rates? If so was this by accident or design?

[Steven Gaal]

• BPA everywhere ,you dont need to be in factory. below
  
• However, in the current study Li argues that the chemical didn’t only have a detrimental effect on individuals with sky-high BPA levels. Even participants with low levels, comparable to the average American, had diminished sperm quality and count. (miss this quote ??)
  
• your theory is that there is a plot to reduce the birth rates in poor ones.// end Colby
  
• NO they want population down in all countries,though more in poorer ones (yes). A technocratic and militaristic class above a slave class would be needed to service elites purposes.
  
• ==============================================
  
• Journal of Human Reproductive Sciences
  SEEMS PROMINET TO ME ??
  

The Journal of Human Reproductive Sciences is a peer-reviewed open-access medical journal published on behalf of the Indian Society of Assisted Reproduction. The journal publishes articles on the subject of assisted conception, endocrinology, physiology and pathology, implantation, and preimplantation genetic diagnosis.

Abstracting and indexing

The journal is indexed with Abstracts on Hygiene and Communicable Diseases, CAB Abstracts, EBSCO databases, Excerpta Medica/EMBASE, and PubMedCentral

GEE YA THINK INDIA KNOWS ABOUT BIRTH CONTROL ?? wiki

[Len Colby]

BPA everywhere ,you dont need to be in factory. below

However, in the current study Li argues that the chemical didn’t only have a detrimental effect on individuals with sky-high BPA levels. Even participants with low levels, comparable to the average American, had diminished sperm quality and count. (miss this quote ??)

Yes supposedly so but according to the author these reductions were proportional to exposure, thus low level exposure presumablly leads to a small reduction.And you still have not produced evidence these leads to lower birth rates or a study confirming Li's findings.

your theory is that there is a plot to reduce the birth rates in poor ones.// end Colby

NO they want population down in all countries,though more in poorer ones (yes). A technocratic and militaristic class above a slave class would be needed to service elites purposes.

==============================================

So do rich men have less exposure to BPA?

Journal of Human Reproductive Sciences

SEEMS PROMINET TO ME ??

The Journal of Human Reproductive Sciences is a peer-reviewed open-access medical journal published on behalf of the Indian Society of Assisted Reproduction. The journal publishes articles on the subject of assisted conception, endocrinology, physiology and pathology, implantation, and preimplantation genetic diagnosis.

Abstracting and indexing

The journal is indexed with Abstracts on Hygiene and Communicable Diseases, CAB Abstracts, EBSCO databases, Excerpta Medica/EMBASE, and PubMedCentral

They are open access (a bad sign) and have an exceptionally low impact factor:

http://www.scimagojr...tip=sid&clean=0

GEE YA THINK INDIA KNOWS ABOUT BIRTH CONTROL ?? wiki

Considering its very high birth rate in the face of extreme poverty and overpopulation not nearly enough.

[Steven Gaal]

Crypto-Eugenic Media Group: Population Control Brainwashing A “Smash Hit”

December 31, 2012

December 28, 2012President of the Population Media Center, Bill Ryerson, recently bragged that its population control brainwashing campaign in developing nations has been “a smash hit.”

The Population Media Center is an American population group producing fictional programs for worldwide dissimination purposes. Its mission: to spread the message of population reduction through works of fiction. Ryerson, the Center’s president, also heads the Washington-based Population Institute. Both groups are active in the modern day “population movement”, striving for worldwide population reduction in the name of the earth.

During an interview with PeakProsperity’s Chris Martenson, Ryerson boasted that (radio) soap-opera’s are the most effective way to cull populations worldwide.

“One of the things that we do – and that is the primary thing we do – is to use a strategy of communications that has turned out, from everything we have been able to measure, to be the most cost-effective strategy for changing behavior with regard to family size and contraceptive use on a per-behavior change basis of any strategy we have found on the planet.”

Ryserson explains that fictional platforms have proven to be successful carriers of the crypto-eugenic message:

“(…) the use of long-running serialized dramas, melodramas like soap operas, in which characters gradually evolve from the middle of the road in that society into positive role models for daughter education, delaying marriage and childbearing until adulthood, spacing of children, limiting of family size, and various other health and social goals of each country. And we have now done such programs in forty-five countries.”

Ryerson continues to brag about the Center’s propaganda campaigns, especially those in Nigeria:

“(…) a program we ran from 2007 to 2009 was listened to by 70% of the population at least weekly. It was a twice a week program. It was clearly a smash hit. And it was a smash hit because it was highly suspenseful and highly entertaining. But it had a storyline dealing with a couple deciding to use family planning, which is almost taboo in northern Nigeria because less than 10% of the people in that region use any modern method of contraception. We had eleven clinics have healthcare workers ask clients what had motivated them to come in for family planning, and 67% percent of them named the program as the motivation.”

“That”, Ryerson explained, “is the kind of thing that can dramatically change demographic trends globally. We need to greatly expand this type of work.”

The Center is heavily sponsored by the corporate and “philanthropic” elites. On its slick-looking website, the Center states “contributions from and partnerships with the following organizations have enabled us to do work which initiates improved health in people all around the world, while providing engaging entertainment!”. Their list counts many UN organizations among its “contributors” as well as USAID divisions operating in so-called “high-fertility nations”:

“The Population Institute, Population Matters, PSI, RAES, Rare, Rollins School of Public Health at Emory University, Save the Children Norway/ Ethiopia, Save the Children US, Texas A&M University, Tree Aid, UCLA School of Public Health, UNDP Burkina Faso, UNDP Headquarters, UNDP Kyrgyzstan, UNDP Papua New Guinea, UNDP Swaziland, UNDP Sierra Leone, UNFPA Burkina Faso, UNFPA Ethiopia, UNFPA Headquarters, UNFPA Jamaica, UNFPA Kyrgyzstan, UNFPA Malawi, UNFPA Nigeria, UNFPA Papua New Guinea, UNFPA Philippines, UNFPA Rwanda, UNFPA Senegal, UNFPA Sierra Leone, UNFPA Vietnam, UNICEF Ethiopia, UNICEF Headquarters, UNICEF Kyrgyzstan, UNICEF Sierra Leone, The Unitarian Society, University Research Co. LLC, UN Women, USAID Jamaica, USAID Kyrgyzstan, USAID Mali, USAID Niger, USAID Rwanda, USAID Senegal, US Embassy/Department of State, U.S. Fish & Wildlife Service.”

In addition, the Center receives funding from literally all the major foundations including the Academy of Television Arts & Sciences Foundation, Bank of America Charitable Gift Fund, The David Rockefeller Fund, Ford Foundation, and the IMF Employees’ Fund.

When we take a look at the Center’s program advisory board we encounter some familiar names, including Paul and Anne Ehrlich, as well as World Bank luminary Herman Daly (who recently called for smaller, less “resource-intensive” people top bred into existence). Interestingly, some names from Hollywood pop up, advising the population center on how to best convince audiences worldwide to cull themselves. Among the names mentioned we find director Jeremy Kagan, renound because of his many film and television projects.

These population reduction propaganda efforts have been going on for a good while. A 1973 UNESCO-publication called Mass Media, Family Planning and Development: Country Case Studies on Media Strategy proposes the creation of “family planning communication resource units” for every nation concerned within the UN-system, especially third-world nations. The reason being, so the report states, that “culturally, there is an emphasis on fertility, and the birth of children to the family is celebrated, as a symbol of prosperity and for status for women.”

“A Communication Resource Unit”, the document reads, “is responsible for the implementation of media policy for one, or more than one field.” The document proceeds with outlining the functions of such a unit in regards to family planning messages:

“The integration of messages is a matter which concerns the Communication Resource Unit, in that an integrated approach to family planning needs to be worked out. (…) These (messages) may be ‘family planning for maternal health’, ‘family planning for family prosperity’, ‘family planning for your figure’, ‘family planning for national prosperity’, family planning for child development.’ These messages will be pretested to find those which seem to appeal most to the eligible age groups.”

conveying the message to unsuspecting audiences: “(…) There are two ways in which the family planning message can be included in feature films. The first is for the family organisation to commission a film specifically for the campaign. (…) if it is to be successful, well known and popular actors must be chosen, and the scripting and direction has to be professionally executed. Another method is for the family planning theme to be introduced into feature films which are already planned and prepared by local commercial production companies. In this case, the family planning organisers must be aware of the possible ways in which the theme can be subtly incorporated, as producers are not likely to respond to a suggestion which involves the total re-thinking of the plot. (…) Suitable opportunities can be found in love stories, in stories based on conflicts between men and women (…).”

As it turns out, these subtle media tactics have indeed proven extremely effective- or, as Ryerson puts it, “a smash hit.”

You might like:

[Steven Gaal]

Saturday, January 5, 2013

Bisphenol A (BPA) Harms Human Fertility Harvard Study Concludes

Activist Post

As human fertility continues to decline at alarming rates around the world, scientists have been trying to pinpoint the cause. A new study may provide the illusive reason why so many are experiencing damaged fertility.

Scientists from Harvard claim they have discovered the "mechanism by which the chemical compound Bisphenol A, commonly used in the plastics industry, damages human eggs and can harm female fertility."

Due to Bisphenol A's (BPA) negative reproductive effects on animals, the scientists decided to test whether human eggs would also be damaged when exposed to BPA.

The results of the Harvard study supported by the National Institutes of Health in the United States and by the Environmental Health Fund in Israel were conclusive.

The results showed that after 30 hours of exposure to Bisphenol A, a greater number of eggs did not mature or began to degenerate. Examination of the chromosomes in the egg showed damage that made proper maturation impossible.

In the group of eggs that were exposed to 20 nanograms per milliliter of Bisphenol A, which is 10 times the average concentration revealed by urine tests of healthy subjects, the rate of eggs that matured and could be used for fertilization was 52 percent, compared to 59 percent in the control group.

In another group of eggs, which were exposed to a quantity of Bisphenol A 100 times the norm, usable eggs declined to 42 percent. When the concentration of the substance was 10,000 times the norm, only 19 percent of the eggs matured. (<a href="

http://www.haaretz.com/news/national/study-chemical-commonly-found-in-plastics-could-harm-fertility.premium-1.491803">

Source

)

“The assumption is that the granulosa cells that surround the egg already contain a basic minuscule amount of Bisphenol A, and we found that continued exposure to concentrations of the substance can harm the supply of eggs and the process of their maturation and female fertility,” said Dr. Ronit Machtinger, a gynecologist who headed the study.

“At present the significance of the findings is that long-term exposure to Bisphenol A, even in a low concentration, has possible effects on fertility and should be avoided, for example, by not heating up food in plastic containers in the microwave,” she recommended.

However, Bisphenol A is not just found in plastics. Significant amounts of BPA can be found in canned goods like soups, tomatoes and soft drinks. It is also found in white dental fillings and nearly all paper receipts that can transferred by touch.

This is not the first study to show that Bisphenol A harms human fertility. In September 2011, a study published in the Journal of Human Reproduction showed that women with high concentrations of BPA trying to conceive children through in vitro fertilization had 24% fewer eggs than women with less BPA present.

The Food and Drug Administration (FDA) is fully aware of the damaging effects of BPA, but to date has only banned baby bottles and sippy cups containing the chemical

[Len Colby]

It seems that Steve has stumbled upon a legitimate issue, BPA probably should be banned or at least better researched/regulated.

Lacking however is any evidence it has reduced birth rates, let alone that it is part of some fiendish plot.

[Steven Gaal]

Studies have also suggested that BPA is linked to other health hazards including increased cancer incidence and premature puberty in children (1). Unfortunately, because of its usefulness, BPA remains as a component in plastic manufacturing. This is especially true in third world countries, where the use of BPA makes plastics production fast and efficient. Thankfully, measures are being taken to curb BPA use. Canada has recently named it a “toxic chemical” and does not allow its use in manufacturing (1). Although its use is being mediated, the vigilant consumer would do well to take heed of labels and ingredient lists to make sure that they are not accidentally consuming this detrimental compound.

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Scientist: Top Selling Weed Killer – Monsanto’s Roundup – Linked to Infertility

http://beforeitsnews.com/health/2013/01/scientist-top-selling-weed-killer-monsantos-roundup-linked-to-infertility-2465144.html

Source: Lisa Garber, Natural Society

Monsanto has made a pretty penny over the last several decades thanks to its top-selling herbicide, Roundup, and cash crops genetically modified to withstand heavy doses of the chemical, such as Roundup Ready corn, soy, and cotton. Meanwhile, Roundup’s key ingredient glyphosate has ravaged the earth, our food chain, and our bodies, and is even causing infertility among the masses. Its effects haven’t gone unnoticed.

Purdue University professor emeritus Dr. Don Huber addressed the dangers of glyphosate to American and European officials in 2011. His concerns centered on an electron microscopic pathogen which “appears to significantly impact the health of plants, animals, and probably human beings.” In a letter to US Department of Agriculture Secretary Tom Vilsack, Huber added that preliminary experiments have reproduced the pathogen’s link to miscarriages.

Vilsack purportedly received Huber’s letter and, despite the available mountain of evidence just one click away, encouraged of Huber “submission of any data or studies in support of his concerns.” Apparently, none of the scientific, published evidence of glyphosate-induced infertility, sterility, birth defects, Parkinson’s, obesity, superweeds, and water pollution were convincing enough to keep the USDA from snuggling down next to Monsanto on a bed of dirty money just 11 days later. Despite Huber’s explicit pleas to delay deregulation of Roundup Ready crops—which would be, in his words, “a calamity”—the USDA went through with deregulation.

This was neither the first nor would it be the last hand members of the government would join with dirty industries to the detriment of their own citizenry. Reuters reports on the myriad concerns raised by numerous members of the scientific and medical community, yet it is only in 2015 that the Environmental Protection Agency promises results for their research into the possible dangers of glyphosate and what, if any, role government should play in it.

Seeing as the government has been front and center in support of Monsanto’s environmentally disastrous antics, we hope to see matched enthusiasm in remedial measures for the environment and its people.

Edited January 12, 2013 by Steven Gaal

[Len Colby]

Studies have also suggested that BPA is linked to other health hazards including increased cancer incidence and premature puberty in children (1). Unfortunately, because of its usefulness, BPA remains as a component in plastic manufacturing. This is especially true in third world countries, where the use of BPA makes plastics production fast and efficient. Thankfully, measures are being taken to curb BPA use. Canada has recently named it a “toxic chemical” and does not allow its use in manufacturing (1). Although its use is being mediated, the vigilant consumer would do well to take heed of labels and ingredient lists to make sure that they are not accidentally consuming this detrimental compound.

This comes from the Dartmouth Undergraduate Journal of Science, a respectable source but it's still just a page/article from an unidentified undergrad, we don't even know his/her area of expertise, there was no citation for this claim. And still have not surmounted problems from previous posts:

Li said such problems were proportionate to exposure, so moderate exposure would only lead to moderate problems.

You've yet to post any evidence slightly – moderately reduced sperm counts lead to reduced birth rates let alone to significant reductions.

You've yet to produce solid evidence that using products made from BPA leads to BPA being absorbed by the organism.

You've yet to produce evidence this part of some plot to harm people's health rather than businessmen putting profits before product safety

+++++++++++++++++++++++++++++++++++++++++++++++++++++++++

Scientist: Top Selling Weed Killer – Monsanto’s Roundup – Linked to Infertility

http://beforeitsnews.com/health/2013/01/scientist-top-selling-weed-killer-monsantos-roundup-linked-to-infertility-2465144.html

Source: Lisa Garber, Natural Society

Huber has been discredited by his own university*. All the other important citations on the page are to other pages on the same or similar sites which in turn link to other pages on the same or similar sites which in turn link to...But let's say it's true as with the above, "You've yet to produce evidence this part of some plot to harm people's health rather than businessmen putting profits before product safety" or that it has reduced birth rates.

* http://www.btny.purdue.edu/weedscience/2011/GlyphosatesImpact11.html

http://pesticidetruths.com/2011/03/02/purdue-university-discredits-own-professor-environmentalist-col-ret-don-m-huber-glyphosate-animals-and-miscarriages-no-scientific-data-fearmongering/

[Steven Gaal]

Made in Britain: The toxic lead used in fuel sold to world's poorest

Source: Independent

A British company convicted of bribing foreign officials to maintain sales of a poisonous lead fuel additive is continuing to sell the chemical abroad to unstable countries, despite mounting evidence that it is responsible for long- term damage to human health and may be linked to violent crime.

Environmental groups today called on the Government to ban Innospec Ltd, which claims to be the world's only producer of tetraethyl lead (TEL), from further exports of the substance. TEL is banned from use on Britain's roads but remains legal in six impoverished nations. The company, which is American owned but maintains much of its manufacturing in the UK, had intended to stop production and sales of TEL at the end of 2012 but has now set a new deadline of the end of this year to halt all dealings in the chemical, from which it has generated large profits.

It recently told shareholders it would seek to "maximise the cash flow" from its declining sales of TEL. The revelations come amid amid renewed focus on the long-term effects of lead pollution following scientific research suggesting an extraordinary correlation between environment between deposits of the heavy metal in the environment, due to leaded fuel and paint, and levels of violent crime in cities.

The US magazine Mother Jones this week highlighted studies which show an apparent link between the rise in leaded petrol use until the 1970s and a spike in violence, with a 20-year gap reflecting the time for children damaged by the metal – including negative effects on the nervous system and IQ – to reach adulthood.

Read more: http://www.independent.co.uk/news/uk/home-news/made-in-britain-the-toxic-lead-used-in-fuel-sold-to-worlds-poorest-8449967.html

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BBC NEWS | Health |

Lead linked to male infertility

news.bbc.co.uk/2/hi/health/2721669.stmCached

Feb 6, 2003 – Men who are exposed to high levels of lead may be at increased risk of becoming infertile, a study suggests.

[Len Colby]

Get back to us with evidence this is part of a conspiracy to depopulate the planet rather than an ethical company putting profits ahead of people's lives.

[Steven Gaal]

SEE AT 10:00 then below article

=========================================================================

Clin Immunol. 2011 Nov;141(2):161-8. doi: 10.1016/j.clim.2011.07.004. Epub 2011 Jul 30.

Cell-mediated immune responses to inactivated trivalent influenza-vaccination are decreased in patients with common variable immunodeficiency.

van Assen S, de Haan A, Holvast A, Horst G, Gorter L, Westra J, Kallenberg CG, Telgt DS, Palache AM, Giezeman KM, Bijl M.

Source

Department of Internal Medicine, Division of Infectious Diseases, University Medical Center Groningen, The Netherlands. s.van.assen@int.umcg.nl

Abstract

Influenza-specific cell-mediated immune (CMI) responses can protect from influenza, but may be decreased in CVID-patients since defects in CMI responses have been demonstrated in CVID-patients. Therefore CMI responses were evaluated in 15 CVID-patients and 15 matched healthy controls (HC) by determining frequencies of interferon (IFN)γ-producing PBMC, and frequencies of IFNγ-, interleukin (IL)-2- and tumour necrosis factor (TNF)α-producing CD4+ and CD8+ T-cells before and after influenza vaccination using IFNγ enzyme-linked immunospot (IFNγ-ELISpot) and flow cytometry. Humoral responses were determined using haemagglutination inhibition assay. In CVID-patients the number of spotforming PBMC in the IFNγ-ELISpot did not increase following influenza vaccination, in contrast to HC. In flow cytometry, the frequencies of IFNγ-producing T-cells decreased in CVID-patients after influenza vaccination, while in HC the frequencies of IFNγ-production flow cytometry increased. Concluding, CMI responses following influenza vaccination are hampered in CVID-patients compared to HC. Additional protective strategies against influenza other than vaccination are warranted.

Copyright © 2011 Elsevier Inc. All rights reserved.

[Len Colby]

Still no evidence this part of some plot and the study concerned "CVID-patients"

http://en.wikipedia.org/wiki/Common_variable_immunodeficiency